Download Current Cancer Research 1995 by Prof. Dr. Dres. h.c. Harald zur Hausen (auth.) PDF

By Prof. Dr. Dres. h.c. Harald zur Hausen (auth.)

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The function of a protein is ultimately dependent on its three-dimensional 52 shape, or conformation, which is in turn genetically determined by the sequence in which its building blocks, the amino acids, are strung together. Phosphorylation changes this shape as well as the proteins's charge. In this way the function of the protein is altered. The direct consequences of phosphorylation can be propagated right through the affected protein and alter its interactions with other macromolecules or even its solubility within the cell.

The Signal then initiates or suppresses the activity of the particular gene. The regulatory sequences within the promoter region function by binding specific proteins called "transcription factors". , suppressed, by the respective promoter. A vast array of regulatory sequences and transcription factors exists in the cell. Different combinations and arrangements of individual DNA elements identified by different transcription factors ultimately result in a chronologically coordinated, individual transcription program for each gene.

Breast cancers presenting a loss of acatenin show diffuse growth without the formation of cell-to-cell contact sites between the carcinoma cells. On the basis of the expression of classical cadherins carcinomas can be classified in four groups: in addition to cancers with a normal cadherin distribution pattern there are cancers with locally reduced cadherin formation. Complete cadherin loss and the formation of aberrant, nonfunctioning cadherin molecules lead to a loss of cell adhesion. This, in turn, results in undifferentiated cell morphology, which means that originally columnar ephitelium cells take on the shape of spindles.

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