Download Endocrine Emergencies: Recognition and Treatment by Lynn Loriaux M.D., Ph.D. (auth.), Lynn Loriaux (eds.) PDF

By Lynn Loriaux M.D., Ph.D. (auth.), Lynn Loriaux (eds.)

Endocrine Emergencies: reputation and therapy offers a state of the art replace at the administration of endocrine, diabetic, and metabolic emergencies. built via popular specialists, this complete and straightforward to learn identify brings the sector totally modern, atmosphere a excessive typical for analysis and remedy in each one class. All chapters commence with a precis that offers, in targeted shape, what the general practitioner must comprehend to start the overview and emergency remedy of the recognized endocrine emergencies. this can be by way of a longer dialogue of the pathophysiology that may be learn after preliminary remedy has began. overlaying such components as hypoglycemia, acute adrenal insufficiency, ovarian hyperstimulation syndrome, thyroid hurricane, and pituitary apoplexy, simply to identify numerous, Endocrine Emergencies: popularity and remedy is aninvaluable, useful source that would be ofgreat curiosity to endocrinologists, inner drugs and emergency room physicians, fellows and residents.

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1993;76(5):1192–8. 2 Diabetic Ketoacidosis and Hyperosmolar Hyperglycemic Syndrome 31 10. Winter SD, Pearson JR, Gabow PA, Schultz AL, Lepoff RB. The fall of the serum anion gap. Arch Intern Med. 1990;150(2):311–3. 11. Sadjadi SA. A new range for the anion gap. Ann Intern Med. 1995;123(10):807. 12. Hypernatremic and polyuric states. ), Newyork, Raven, 1992, p. 1578. 13. Yadav D, Nair S, Norkus EP, Pitchumoni CS. Nonspecific hyperamylasemia and hyperlipasemia in diabetic ketoacidosis: incidence and correlation with biochemical abnormalities.

This recommendation is to protect the plasma volume; once insulin is administered, the consequent fall in circulating glucose concentrations will lead to an intracellular shift of water from the plasma compartment which can result in a precipitous drop in systemic blood pressure. 1 U/kg body weight/hour continuous infusion. The goal of insulin therapy is to decrease serum glucose by 50–75 mg/dL/h. Overly aggressive reduction of glucose may result in brain edema. 5. Glucose levels should be monitored every 1 h initially, and once stabilized, every 2–3 h.

Wiethop BV, Cryer PE. Alanine and terbutaline in treatment of hypoglycemia in IDDM. Diabetes Care. 1993;16:1131–6. 6. Gunning RR, Garber AJ. Bioactivity of instant glucose. Failure of absorption through oral mucosa. JAMA. 1978;240:1611–2. 7. Haymond MW, Schreiner B. Mini-dose glucagon rescue for hypoglycemia in children with type 1 diabetes. Diabetes Care. 2001;24:643–5. 8. Basu R, Basu A, Johnson CM, Schwenk WF, Rizza RA. Insulin dose–response curves for stimulation of splanchnic glucose uptake and suppression of endogenous glucose production differ in nondiabetic humans and are abnormal in people with type 2 diabetes.

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